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CEBPG suppresses ferroptosis through transcriptional control of SLC7A11 in ovarian cancer

Consistently, in the GLS-deficient mouse tumor model, the increased concentration of glutamine in the TME due to restricted glutamine utilization by tumor cells leads to elevated levels of glutamine available to tumor-infiltrating T lymphocytes, thereby enhancing its anti-tumor activity [17]

Most homocysteine is remethylated to methionine using methyl groups from the folate cycle or donated from trimethylglycine (also called betaine)

The rate limiting amino acid in de novo synthesis of GSH in metabolic disease appears to be glycine