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Evidence for UA neuroprotection in neurodegenerative diseases In 1955, Orowan et al

Metabolic reprogramming driven by oncogenes allows cancer cells to maintain deregulated cell proliferation, withstand metabolic challenges that are associated with diminished oxygen and nutrients, maintain a dedifferentiated state through alterations in global patterns of gene expression, and corrupt the surrounding microenvironment to actively facilitate tumor growth and dissemination 16,17,18

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If the individual has one copy of each mutation (AC) he/she is said to have double heterozygous mutation