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The peptide's glucose-lowering effects partially result from its ability to increase fatty acid oxidation in muscle tissue, reducing free fatty acid flux to the liver and improving insulin's suppression of hepatic glucose output

This is a well-established feature of GPCR biology

Hydrophilic NPs will also behave differently to those with hydrophobic surfaces [49, 123]

Navrot et al., 2006), but they generally lack the selenocysteine (SeCys) residue critical for the high catalytic efficiency of animal GPXs, substituting it with cysteine ( Figure 7 ) (Herbette et al., 2007