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Free Radical Biology and Medicine 46, 443-453CrossRefGoogle ScholarPubMed 74Chanas, S.A., Jiang, Q., McMahon, M

At this exposure age, non-genomic estrogenic pathways associated with apoptosis were activated (Fig

FACS analysis showed decreased trend in the production of VEGF, CXCL8 by three PCa and the BPH cell lines exposed to ALCAR 1 and 10 mM, following 24 h of treatment (Fig