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HDAC9 knockout mice exhibited normal baseline synaptic transmission but failed to maintain hippocampal LTP, a cellular correlate of learning and memory, indicating impaired synaptic plasticity

Mature Np effectively degrades gliadin and the 33-mer in vitro, and preliminary studies in standard C57BL/6 mice have shown that co-administering gliadin with minute amounts of the Np zymogen, pro-neprosin (pNp), significantly reduces GIP levels in the small intestine (del Amo-Maestro et al, 2022)

The dysregulation of cell death and defective clearance of dying cells are closely associated with the pathogenesis of LN [131]

a lower concentration gives more flexibility in fine-tuning small volumes