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This finding correlates with those described by Kumar et al., who demonstrated that ER is crucial to enhance OLs production, myelin protein synthesis, remyelination, and axon conduction in EAE mice through the activation of the PI3K/Akt/mTOR signaling pathway, without disturbing inflammation in the corpus callosum or spinal cord (Kumar et al., 2013

Some patients primarily exhibit elevated TSPO signals, indicative of an inflammation-driven phenotype, while others show reduced FDG metabolism, characteristic of a metabolic defect-driven phenotype, with no direct correlation between the two (147)

Lee, J.S

Tailor Made Compounding LLCs case illustrates this principle