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Abstract Superoxide dismutase-1 (SOD1) mutants, including those with unaltered enzymatic activity, are known to cause amyotrophic lateral sclerosis (ALS)

In this context, DHODH, a mitochondrial inner-membrane enzyme traditionally known for its role in de novo pyrimidine biosynthesis, was proposed to also function as a CoQ 10 oxidoreductase and ferroptosis suppressor

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Despite advances in antimicrobial therapy and supportive care, sepsis continues to result in high morbidity and mortality, with an estimated 48.9 million cases and 11 million deaths worldwide in 2017, accounting for nearly 20% of all global mortality, disproportionately affecting low- and middle-income countries (Rudd et al., 2020)