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Controlled human trials specifically evaluating AOD-9604 for joint pain and osteoarthritis are needed to establish the level of efficacy and optimal treatment protocols

The two peptides promote tissue repair and regeneration through distinct and largely non-overlapping molecular mechanisms that converge on shared biological endpoints, providing the basis for additive or synergistic effects when both are present in the same research model

This is proposed to be mediated by TRX1 oxidation by hydrogen peroxide which causes dissociation of ASK1 thereby triggering anti-apoptotic factors via downstream activation of MAPK kinase MKK3/MKK6/p38 and MKK4/MKK7/JNK pathways (Wagner et al