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Patients can present with widespread punctate lesions or surface erosions of the cornea, as well as persistent detachment and epithelial damage, and inflammatory reactions, ultimately endangering vision (148, 149)

In certain lung cancer cellular models, it has become well established that the pro-oncogenic role of PRDX1 is mediated by the activation of c-Jun and AP-1 (70), and that the transforming growth factor 1 (TGF1)-induced EMT is caused by direct inhibition of E-cadherin expression (71)

Organic methylmercury intoxication results in substantial neuronal depletion and gliosis in the calcarine and parietal cortices of the brain and cerebellar folia, as was demonstrated in patients presenting with classic Minamata disease

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