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Its enzymatic function is based on two aspects: its catalytic activity and the auto- S -glutathionylation of GST by itself on Cys47 and Cys10, both of which disturb the subsequent interaction with c-Jun NH 2 -terminal kinase (JNK), resulting in the formation of a GST multimer 30 (Figure 3)

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In addition, Fer-1 alleviated DN and ameliorated renal hypertrophy and albuminuria by inhibiting ferroptosis, and ultimately reduced the accumulation of intracranial lipid peroxides in diabetic mice through the HIF-1/HO-1 pathway [242]