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The loss of function of E-cadherin is associated with cancer progression by enhancing proliferation, invasion, and metastasis, and the shift toward increased expression of N-cadherin contributes to the disruption of intercellular junctions and initiates the process of epithelialmesenchymal transition (Jiang et al

In the nervous system, some nerve cells regulated the immune response and cell repair

Mitochondrial dysfunction in tumors is highly heterogeneous, influencing disease phenotypes and treatment responses across cancer types