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M., Aksenova M., Aksenov M., Butterfield D

In these assays, macrophages are strongly driven towards the M2 phenotype by the presence of the potent cytokines IL-10 and M-CSF

This has not been demonstrated in clinical practice with oral glutathione at standard supplemental doses, but preclinical evidence warrants caution in cancer patients undergoing active treatment [2][5]

In human monocytes and macrophages, the inhibition of the autophagy pathway drives the formation of the NLRP3 inflammasome, while increased autophagic flux reduces the secretion of IL-1, suggesting the role of autophagy impairment in driving inflammasome activation [192]