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Interestingly, the expression of the nitric oxide synthases, eNOS and iNOS, involved in NO production and hemodynamic changes was also altered in Gstp1/2 / mice

Thats where cellular repair and antioxidant support comes in

Iron overload is another factor to promote tissue oxidation during ischemia and reperfusion, and becomes a novel risk factor for predicting hepatic I/R injury in liver transplantation and lung oxidative injury with ischemia/reperfusion in rat lungs [18, 92,93,94]