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No form is universally superior, and clinical data is still evolving

[4] [5] Readers should note that these figures reflect specific trial protocols and may not represent the doses used in ongoing phase 3 studies or any future licensed product

Successively, GSH was also found to be a powerful inhibitor of other viruses including herpes simplex virus type 1 (HSV-1), Sendai and influenza

455 Their role in targeting GBM angiogenesis has also been explored, with miRNA-29a-3p-enriched MSC-derived EVs suppressing vasculogenic mimicry and angiogenesis independently of VEGF, suggesting a promising antiangiogenic strategy