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doi: 59

British Journal of Clinical Pharmacology, 48(3), 424-430

EC acts both directlyvia suppression of factor-kappa B (NF-B), mitogen-activated protein kinase (MAPK), and Janus kinase/signal transducers and activators of transcription (JAK/STAT) pathwaysand indirectly through activation of nuclear factor erythroid 2-related factor 2 (Nrf2)-driven antioxidant responses that mitigate inflammatory damage