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The first hypothesis outlined in the Introduction assumes that the AP activity in the trxB mutant is a result of the failure to reduce AP due to the absence of the reduced forms of the two thioredoxins

We here evaluated the changes in the cytotoxic potential of liposomes encapsulated with stigmasterol and its esters having undergone gastrointestinal digestion simulated in the artificial digestive tract

Both clinical and experimental data have increasingly implicated neuroinflammation as a key contributor to visceral hypersensitivity (138), with bidirectional nociceptor-EGC communication initiating and amplifying pro-inflammatory cascades (139)