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The lactate accumulation from aerobic glycolysis acidifies the TME and promotes the M2 tumor-associated macrophage (TAM) polarization of these macrophages and microglial cells

Interestingly, mIECs lacking Stard7 critically rely on fatty acid oxidation (FAO) as the maximal oxygen consumption rate (OCR) dropped in Stard7-depleted but not in control cells when treated with Etomoxir, a FAO inhibitor (Fig

W43 of Affimer K6, and V7/L56 from KRAS form a hydrophobic cluster strengthened by Affimer residues T41 and Q45 forming hydrogen bonds with KRAS switch region residues D38/S39 and Y71, respectively (Fig