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Cell death signaling pathways in ischemic stroke Damage caused by excitotoxicity, oxidative stress, and mitochondrial dysfunctions in ischemic stroke may induce a variety of cellular signaling cascades, which lead neural cells to undergo either programmed or unprogrammed death

Such discrepancies point out to possible missing links in the metabolic pathways based on the current status of the gene sequencing and annotations

The molecular formula and weight for nucleic acid (Supplementary Table 1) was estimated using the GC (guanine-cytosine) content of Synechococcus elongatus (60.67%) 89 for DNA and Escherichia coli for RNA (50.8%) 90