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As a standalone therapy, it shows promise in treating early-stage cancers by targeting critical hallmarks such as proliferation, apoptosis, angiogenesis, and metastasis

Reports that newborns with a complete lack of insulin effects due to inactivating defects in the INSR gene can now survive for extended time into adolescence when treated with recombinant IGF-I, that stimulate glucose uptake via the IGF1R sharing common signaling pathways with the INSR, support an important direct role of IGF-1 signaling on metabolism (93)

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