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Interestingly, GSK-3 was reported as a positive regulator of ferroptosis

Dehydroascorbate (DHA), an oxidant, is formed from ascorbic acid during the removal of ROS, yet the enzyme responsible for converting DHA back to ascorbic acid is dependent on GSH

This is the most speculative of the three pathways - direct human evidence is limited - but it is the rationale most often given for pairing MOTS-c with retatrutide specifically rather than with a slower-deficit compound