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Their characterization by electrophysiological approaches revealed that they are mostly closed under basal conditions and activated by different stimuli that are also known to induce eryptosis: ROS, exposure to high extracellular osmolarity, or removal of extracellular Cl - and isosmotic replacement with gluconate activate the channel [98] and trigger eryptosis [90]

Chronic heart failure is closely linked to mitochondrial dysfunction, primarily caused by cumulative damage from high-level reactive oxygen metabolism (Sanada et al., 2011)

doi:10.1002/14651858.CD007348.pub3

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