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231 Mutation of CTNNB1 exon 3 (87.0%

used a transgenic approach to carry out similar gain- and loss-of-function experiments in the developing mouse cochlea in vivo (Shi et al., 2014)

The Benoist 2014 study used the HGF/MET dimerization antagonist Hinge to block Dihexas procognitive activity in scopolamine-impaired rats, with oral Dihexa (2 mg/kg/day) reversing the scopolamine-induced Morris water maze deficit when administered alone but losing effect when co-administered with Hinge [3]