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Identification of a novel chemokine-dependent molecular mechanism underlying rheumatoid arthritis-associated autoantibody-mediated bone loss

It is studied for its ability to interact with dysregulated cellular pathways commonly found in transformed cells, particularly involving the MDM2 regulatory system

The spent PtGaPb catalyst could be regenerated by O 2 treatment to recover the original catalytic performance after some induction period, whereas some other bimetallic or trimetallic Sn-containing catalysts (Pt 3 Sn, PtSn, and PtGaSn) did not (Supplementary Fig

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