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These findings highlight the potential of dual CYP17A1 and HDAC6 inhibition as a promising strategy to overcome resistance in GBM therapy, offering a new avenue for more effective treatments

Luteolin also promotes apoptosis-inducing factor (AIF) nuclear translocation via ERK and p38 activation

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The results show the role of cellcell interaction and intracellular NRF2-YAP signaling pathway in determining ferroptosis and show that malignant mutations in the NRF2-YAP signaling pathway can predict the response of cancer cells to future induction therapy for ferroptosis [72]