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Upon inflammasome activation by nigericin stimulated potassium efflux, we found increased -tubulin acetylation in LPS primed peritoneal macrophages (Fig 4A), BMDMs (Fig

ERAD helps mitigate this burden by degrading these proteins, thereby preventing their aggregation and the subsequent activation of detrimental pathways, such as chronic ER stress, autophagy dysfunction, and apoptotic signaling