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These selective inhibitors are effective anti-inflammatory drugs, and they produce less GI bleeding and ulcers than do the nonselective COX inhibitors

PA most likely prevented activation of the pro-inflammatory factors and reduced LPS/D-Gal-induced liver injury by enhancing the zinc finger E-box-binding homeobox 2 expression level, and consequently, blocking activation of the NF-B signaling pathway in the liver (Yang et al., 2021a)