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Excessive oxidative stress disrupts DG neurogenesis by impairing progenitor proliferation, promoting apoptosis, and inhibiting neuronal differentiation [45-47]

Table 1: In Vitro Protocol - ERK Activation and ROS in Neuronal Cells This protocol is based on studies using rat cerebellar granule cells [2] [3]

The c-jun NH2-terminal kinases (JNKs) are recognized as critical regulators of apoptosis, initiated in reaction to cellular damage

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