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Targeting NLRP3 inflammasome components or Il1r1 strongly inhibited not only VEGFAinduced cataract formation, but also both neovascular and nonexudative AMDlike pathologies

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An insufficient aspartate supply leads to mitochondrial metabolic dysfunction, inhibition of mTOR signaling, and reduced expression of cytotoxic molecules such as granzyme B (GZMB) and IFN-, ultimately impairing T-cell cytotoxicity

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