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Strikingly, these favorable features conferred by QMP were independent of the presence of a C-terminal L585

Microscopically, transmural hyperemia of the gastrointestinal tract, intestinal mucosa villi reduction, crypt reduction with focal denudation of superficial epithelia, and large bowel dilatation were all inhibited

Under hyposalinity, the addition of exogenous GSH and BSO weakened the hyposalinityinduced increase of the endogenous GSH content and ratio of GSH/GSSG ( Figure 1 )

This unique substrate specificity and acid stability make RmuAP1 a promising candidate for oral enzyme therapy to aid gluten digestion in individuals with CD, potentially reducing gluten-induced intestinal damage (Zhang et al