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In myofibroblasts, however, TGF- upregulates GLS1 expression via Smad3 and p38 and promotes myofibroblast differentiation ( Tryptophan metabolism, especially in immune cells, exemplifies another aspect of amino acid metabolism modulated by TGF- signaling (Table 1C)

persici within the host, we first extracted reads that were not mapped to the host genome in the RNA-seq data, and then mapped these reads to the complete A

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GSH depletion will lower a cell's capacity to buffer against endogenous oxidants, and it may set a time limit on continued mitochondrial function and thus indirectly on total ATP levels and membrane integrity