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However, long-term treatment increased the density of oligodendrocytes at the lesion site (Cooper et al., 2024)

In this classic theory, the interaction between Ach and calcium plays a substantial role, with Ach thought to promote intracellular calcium release and mediate calcium influx by activating G protein-coupled receptors, resulting in an increase in intracellular calcium concentration

Rathinam VAK, Jiang Z, Waggoner SN, Sharma S, Cole LE, Waggoner L, et al

Although NADPH-stimulated O 2 - production was enhanced, direct evidence of increased oxidative stress in the unstimulated myocardium was not shown, suggesting the possibility of compensation from increased O 2 - buffering capacity