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Rapid administration may therefore precipitate acute hypernatremia and osmotic shifts that transiently reduce cerebrospinal fluid pressure, a concern particularly in neonates and patients with traumatic brain injury

At this higher ratio, HGFs binding affinity for c-met increased more than two-fold compared with untreated HGF, while also becoming more resistant to nitration-induced dysfunction, especially at Y198

doi: 10.1016/j.trecan.2021.02.004 Summary Keywords metabolic reprogramming, phosphoglycerate dehydrogenase (PHGDH), serine metabolism, serine synthesis pathway (SSP), targeted therapy, tumor immune microenvironment Citation Su P, Yang Y and Zheng H (2026) The serine code of metabolic reprogramming: multidimensional roles of the serine synthesis pathway in tumors and novel breakthroughs for targeted therapy