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The mammalian target of rapamycin (mTOR) pathway is involved in the differentiation of oligodendrocyte precursor cells (OPCs), with inhibition of this pathway through rapamycin treatment shown to block OPC differentiation and decrease expression of myelin proteins (Tyler et al., 2009

Zincs anti-inflammatory effects were also linked to inhibition of Notch1 pathway, which regulates M1 macrophage polarization [224]

Successful activation of immune therapy recruits CD8 T cells to the tumor site, enhancing tumor cell ferroptosis via IFN-related pathways, representing a significant advancement in efficient tumor eradication strategies