glutathione in ovarian cancer a double-edged sword

glutathione in ovarian cancer a double-edged sword Targeting ferroptosis cancer: Novel strategies to overcome chemotherapy resistance Metabolic reprogramming of three major

Name: glutathione in ovarian cancer a double-edged sword Targeting ferroptosis cancer: Novel strategies to overcome chemotherapy resistance Metabolic reprogramming of three major
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Metabolic reprogramming of three major nutrients in platinum resistant ovarian cancer PMC Redox Regulated Iron Metabolism and Ferroptosis in Ovarian Cancer: Molecular Insights and Therapeutic Opportunities Cysteine metabolic circuitries: druggable targets in cancer British Journal of Cancer Frontiers Ferroptosis: a dual edged sword in tumour growth

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Compared to healthy controls, SCD patients have significantly lower erythrocyte GSH and cysteine concentrations 12 and a faster rate of GSH synthesis, suggesting that the endogenous cysteine supply is insufficient to meet all anabolic demands

Firstly, we verified across a panel of normal and cancer cell lines with differing p53 status (Supplementary Table 1) that treatment with a lethal dose of APR-246 resulted in higher intracellular ROS (Fig