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Subsequent work by Sun and colleagues in the APP/PS1 mouse model identified downstream PI3K/AKT pathway activation as a major component of the mechanism, with the PI3K inhibitor wortmannin reversing Dihexas anti-inflammatory and anti-apoptotic effects [5]

Structural analysis by the comparative molecular field analysis method of the affinity of -adrenoreceptor blocking agents for 5-HT1A and 5-HT1B receptors