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In our recent study, in addition to elucidating the pathogenic mechanism of FUS-associated ALS in both Drosophila and animal systems, we found that overexpression of GstO2, a Drosophila homolog of human GSTO1, reduces cytoplasmic FUS aggregates and attenuates neurodegenerative phenotypes, including mitochondrial dysfunction and neuronal toxicity 148

The relationship between RT and cuproptosis is also highly significant

(2014) found that a relatively low risk for incidence of cardiovascular disease for those with a greater intake of ascorbic acid supplements

Catalase's capacity to lower oxidative stress and inflammation shows its potential as a therapeutic agent in the treatment of CAP, which might lead to better clinical results and reduced disease severity [95, 96]