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doi: 10.1038/nature04246 3 ApfelbachR.BlanchardC

These data are consistent with the distinct functions of GluN2B containing NMDARs during development and the induction of plasticity, and GluN2A NMDAR heterotetramers in the mature brain and after plasticity 3,9,10,37,38

Validation of Monte Carlo generated phase-space descriptions of medical linear accelerators

Given the shared effectors in inflammation and cancer progression, epigenetic modifications are assuming evergreater significance in deciphering inflammation, cancer, and the interactions between them