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PARPs-induced ADP-ribose marks elevate 10- to 27-fold in response to the oxidative genome damage by H 2 O 2 in human osteosarcoma cells

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This pathway activation enables: Enhanced cell adhesion to extracellular matrix components Increased migration of repair cells to injury sites Improved cell survival under oxidative stress conditions Accelerated tissue remodeling and wound closure Studies using fluorescent staining revealed that BPC-157 peptide potently stimulates F-actin formation, the structural protein essential for cell movement[1]

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