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This distinction has important clinical implications, as defining a pathogenic role of increased VEGFA signaling in specific aging diseases may suggest novel therapeutic approaches for these diseases that target VEGFA pathway hyperactivation or downstream cellular pathomechanisms that occur as a consequence of increased VEGFA levels

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The increase in Chac1-Myc expression by HA-Ub (WT) co-expression was almost at the same level as observed by HA-Ub (K48)