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They rapidly break inside cells, where the environment is a reducing one due to the high concentration of glutathione (GSH), while the oxidizing extracellular environment provides them long-term stability (Wang et al., 2016)

Commonly encountered drugs that interact with DAAs include antiarrhythmic drugs such as amiodarone, antiepileptic and barbiturate drugs (e.g., carbamazepine, oxcarbazepine, phenobarbital, and phenytoin), antipsychotic medications, antituberculosis drugs (e.g., rifampin and rifapentine), anticoagulant medications, and antidiabetic agents [64]

9.1), and calcitonin, a polypeptide hormone

The FSP1/CoQ10 pathway suppresses lipid peroxidation and ferroptosis by utilizing FSP1 to regenerate reduced CoQ10, a lipophilic radical-trapping agent, via NADPH