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Several authors have already reported the linkage of CAT, GPx and Prx2 to the erythrocyte membrane in case of metabolic stress and/or OS

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demonstrated that the inhibition of glycolysis strongly reduced the early phase of the oxygen consumption rate (as readout of the oxidative burst) and ROS release in mouse bone marrow-derived neutrophils stimulated with PMA, while inhibition of mitochondrial respiration impacted to a small degree late-phase ROS release (Furukawa et al., 2000