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Recently, other pathways including AMPK/ULK1 axis that regulate mitophagy were found [49]

Copy-number amplifications of serine- and glycine-synthesis genes and genetic alterations in common oncogenes and tumour-suppressor genes enhance serine and glycine synthesis, resulting in high production and secretion of these oncogenesis-supportive metabolites

doi:10.1016/j.chembiol.2009.12.011 67

It has been reported, that in addition to mitochondrial sites of ROS formation, several non-mitochondrial sites may be triggered following hyperglycemia