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Homozygous deletion of Querkopf, the mouse homolog of human KAT6B, results in craniofacial abnormalities and defects in central nervous system development in mice [28] , resembling human phenotypes such as growth retardation, facial dysmorphism, and brain anomalies seen in Noonan syndrome [27]

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Moreover, other lines of research indicate that acyl-carnitine supplementation can be considered as a potential therapeutic strategy across several neuropsychiatric disorders (i.e., schizophrenia, autism, depression, etc.) with unique neuroprotective effects including anti-inflammatory and antioxidant benefits 27,43