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We hypothesize that decreased expression of C9 after SB application may reduce post-ischemic brain damage in the early phase after HI, while in the later phase, enhanced expression of this protein may promote repair processes (e.g., by tagging dead neurons to remove from the tissue by microglia)

The post then shows on the calendar as pending approval in yellow, and reviewers get an email with a link to approve or reject

Exogenous AntioxidantsDouble-Edged Swords in Cellular Redox State Health Beneficial Effects at Physiologic Doses versus Deleterious Effects at High Doses

Failure of communication between patients with cerebral aneurysms and neurosurgeons