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& Severinghaus, J

The accumulation of misfolded protein in the endoplasmic reticulum (ER) leads to ER dysfunction, which is known as ER stress ( 12 regulated ER homeostasis ( 12 deficiency activated ER stress pathways by increasing the phosphorylation of PERK and IRE1 and the expression of ATF6 ( 12 could promote nerve regeneration after TBI by regulating ER stress

larger prospective trials are needed to establish the benefit of corticosteroids

Access this article Similar content being viewed by others References Papers of particular interest, published recently, have been highlighted as: Of importance Of major importance Devine CJ Jr, et al