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Inhibition of mitochondrial complex I leading to NAD(+)/NADH imbalance in type 2 diabetic patients who developed late stent thrombosis: evidence from an integrative analysis of platelet bioenergetics and metabolomics

Within the tumor microenvironment (TME), they collectively influence anti-tumor immune responses through metabolic reprogramming, immune cell polarization, and regulation of immune checkpoint expression

The origin of these strictures seems multi-factorial, but damage to the bile duct stem cell niche is found to play an important role

however, METH treatment caused time-dependent reductions in the protein contents of complex IV (Wu et al., 2007)