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It is therefore reasonable to assume that PD could be modulated by differentially targeting the three predominant sirtuins (SIRT1, 2, and 3), which are of primary relevance to mitochondrial dysfunction in PD (Lin et al., 2018)

doi: 10.24976/Discov.Med.202436183.61

On the one hand, four studies demonstrated that prenatal RSV treatment could reverse VPA-induced social deficits ( RSV prevented or ameliorated abnormalities in animal models of ASD through various molecular mechanisms, mainly anti-oxidative stress, and anti-inflammatory functions

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