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Central to the pathogenesis of atherosclerosis are the interactions among monocytes, macrophages, and neutrophils, which play pivotal roles in the initiation, progression, and destabilization of atherosclerotic lesions

The minimal systemic exposure of these potent agents ensures that other organs like the liver, spleen, and kidney are protected from exposure

Other forms include guttate psoriasis, which presents with numerous small, drop-like lesions and often follows streptococcal infection (Leung et al

TABLE 42 Human liver P450s (CYPs), and some of the drugs metabolized (substrates), inducers, and selective inhibitors