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Bax has been suggested to be required for Drp1-mediated mitochondrial fission caused by photodynamic therapy in human lung adenocarcinoma cells [76], while Drp1 influenced Bax translocation to mitochondria in response to irradiation-induced apoptosis [77] and Drp1-induced membrane remodeling stimulates Bax oligomerization [78]

Early studies indicated generally adequate riboflavin (B2) and pyridoxine (B6) levels in treated patients (Congden et al., 1981)

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