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The authors attributed this to the fusion protein being soluble in the E

Mitochondria-targeted catalase reverts the neurotoxicity of h SOD1G(9)(3) A astrocytes without extending the survival of ALS-linked mutant hSOD1 mice

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While BPC-157 research focuses on localised repair mechanisms and growth factor upregulation, TB-500 research centres on cell migration and anti-inflammatory pathways